Monthly Archives: May 2019

Do you get diabetes from eating too much sugar?

I have just returned from 2 weeks in the UK. I attended my nephews wedding, which was held a beautiful Devon town called Budleigh Salterton. My plane was delayed so returned a bit late, and am just getting over jet lag. I am keen to get back to work next week. Dr Nixon and Dr Mahdu kindly looked after my patients while I was away. I put on a few kilos from all the festivities and the plane so will have to get back to healthy habits from today.

Do you get diabetes from eating too much sugar?

There is a widespread belief that sugar is the sole cause of diabetes. After all, the disease is characterised by high levels of sugar in the blood.

Diabetes was first identified through the sweet smell of urine, and it later became apparent that sweet, sugary urine signified a high level of blood sugar. Over time, diabetes treatment has swung from eating primarily sugar (to replace what is lost), to avoiding sweetness (to limit high sugar levels).

Today, the debate on amounts and thresholds of sugar and its role in diabetes seems as fierce as ever.

Type 1 diabetes is an autoimmune disease, where genetics and environmental factors interact. Some research suggests that sugar intake may play a role in the development of type 1 diabetes, but the research isn’t conclusive.

For type 2 diabetes, a diet high in sugar could, in principle, influence or accelerate the progression of the disease depending on the pattern of consumption. But to suggest that dietary sugar might cause or contribute to type 2 diabetes needs strong scientific evidence that demonstrates that either sugar increases body weight and body fatness (necessary for type 2 diabetes), or that sugar has some kind of unique effect that leads to 2 diabetes, irrespective of weight or body fatness.

What we mean when we talk about sugar

What most people understand to be sugar is sucrose: a mix of glucose and fructose. A common misunderstanding is that blood glucose is derived solely from dietary sugar. Almost all of the sugar in the body, including the blood, is in the form of glucose – one of many sugars belonging to the family of carbohydrates.

Sugars typically form a small part of the diet, not all of them are equally effective at increasing blood glucose levels, and other carbohydrates, as well as fats and protein, influence glucose levels, too.

Animal studies show that high sugar diets lead to rapid weight gain and impair the body’s ability to effectively regulate blood glucose. But these effects are mainly due to the fructose component of sucrose and not glucose.

In people, diets high in sugar have also been shown to increase weight as well as risk factors for cardiovascular disease. But these effects only seem to occur when calories are not being controlled; simply exchanging extra sugar with calories from another source won’t prevent these negative effects. Also, observational studies have failed to show a harmful association between dietary sugar and type 2 diabetes.

Type 2 diabetes has arisen through rising body weights. Fatter people eat more of many things – not only sugar – and extra calories from any nutrient will lead to weight gain. Most sugary processed foods, such cakes and chocolate, contain large amounts of fat which contribute heavily to the calorie content.

Type 2 diabetes is linked to rising body weight. Suzanne Tucker/Shutterstock.com

Nothing special about sugar

Recently, the debate has turned to sugar-sweetened drinks, such as fizzy drinks. Sugars in drinks are less satiating than sugars in solid foods, and this may drive our appetite to eat more. Sugary drinks have been linked to type 2 diabetes, independent of body fatness. But so have artificially sweetened low calorie drinks. Fruit juices, though, have not been linked to type 2 diabetes despite having similar sugar contents to fizzy drinks.

There is nothing special about sugar that sets it apart from other foods, and sugar does not cause type 2 diabetes on its own. Generally, people eating lots of sugar tend to have poorer diets and unhealthier lifestyles. These, as well as other factors including urban growth patterns, the built environment, the food environment, stressful jobs, poor sleep and food pricing probably contribute more to the rising incidence of type 2 diabetes than dietary sugar.

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Is apple cider vinegar really a wonder food?

I am going to be away for the next 2 weeks. I am going to my nephew’s wedding in Devon, UK, and will visit some friends while I am there. Dr Anne Nixon will look after any urgent issues that arise while I am gone. I take apple cider Vinegar daily and feel better for it. ?placebo effect – perhaps.

Is apple cider vinegar really a wonder food?

Folk medicine has favoured apple cider vinegar for centuries and many claims are made for its supposed benefits.

Apple cider vinegar is made by chopping apples, covering them with water and leaving them at room temperature until the natural sugars ferment and form ethanol. Bacteria then convert this alcohol into acetic acid.

Strands of a “mother” will form in the cider. These are strained out of many products but left in others, and are often the target of health claims. The “mother” can also be used to start the production of the next batch of cider.

But will apple cider vinegar really help you lose weight, fight heart disease, control blood sugar and prevent cancer? And what about claims it is rich in enzymes and nutrients such as potassium?


Read more: The long, strange history of dieting fads


Weight loss

The evidence that apple cider vinegar helps fight fat is weak.

A short-term study in Japan added two daily drinks of 15 millilitres of apple cider vinegar mixed with 250 ml of water to the usual diet of overweight men and women. Their weight fell by about one kilogram over 12 weeks, but returned to usual levels within four weeks.

According to a UK study, it may be that vinegar can suppress appetite. When offered a pleasant-tasting vinegar drink, one that was less palatable, or a non-vinegar drink with their breakfast, volunteers who downed both vinegar drinks felt slightly nauseated. Not surprisingly, this depressed their appetite, with the least palatable vinegar drink having the greatest effect.

 

The ‘mother’ is strained from some cider vinegars and left in others. Mike Mozart, CC BY

Others claim taking apple cider vinegar with meals will help digest proteins faster and therefore generate higher levels of growth hormone. This is claimed to break down more fat cells. Unfortunately, there’s no evidence to support such ideas.

Claims that pectin – a type of viscous dietary fibre – in cider vinegar will help weight loss by making you feel full for longer ignores the fact that the pectin in apples is not found in apple cider vinegar.

Heart disease

Pectin is again credited for cider vinegar’s supposed benefits for heart disease, with claims it “attracts bad LDL cholesterol”.

However, the Japanese study referred to for weight loss found no difference in LDL cholesterol with either a low or higher amount of cider vinegar over a 12-week period.


Read more – Heart disease: what happens when the ticker wears and tears


Others claim that cider vinegar works like a broom to clean toxic wastes out of the arteries. Sadly, there’s no evidence for that one either.

Blood sugar and diabetes

Several studies have reported on the effects cider vinegar can have on blood glucose levels. One small study of healthy volunteers found that adding vinegar to a meal reduced glucose and insulin levels – at least for 45 minutes – and increased satiety for up to two hours.

Another small study of people with type 2 diabetes reported adding vinegar to a high carbohydrate meal reduced the subsequent rise in the blood glucose level.

However, this effect was only apparent for a high glycaemic index carbohydrate, such as mashed potatoes. When the carbs came from a lower GI food such as wholegrain bread, the vinegar had no effect.


Read more – Explainer: what is diabetes


A word of warning for those with type 1 diabetes who also have damage to the vagus nerve (a common co-problem): when taking apple cider vinegar in water before a carb-rich meal, the delay in the stomach contents passing to the small intestine may alter the quantity of insulin so the usual daily injection may be inappropriate.

Other diseases

As for allergies, acne, arthritis, hiccups and leg cramps, there is no evidence that apple cider vinegar prevents or cures any of these conditions.

Nor is there evidence from any studies that cider vinegar has benefits for preventing or curing cancer. Unproven cancer cures can waste valuable time in seeking reliable treatments.

So is it worth taking?

Some sites promoting unrefined cider vinegar claim it is a good source of potassium. We certainly need potassium to help regulate the balance of water and acidity in the blood.

But with apple cider manufacturers declaring their products have just 11 milligrams per 15 ml serve (and a recommendation for two serves a day) it is a negligible source. The recommended dietary intake of potassium is 2,800 mg/day for women and 3,800 mg/day for men. Bananas have around 400 mg.

 

An average banana has 400 mg of potassium. Scott Webb/Unsplash

In Australia, products cannot claim to be a source of any nutrient unless a reasonable daily intake provides at least 10% of the recommended daily intake (RDI). A “good source” must have 25% of the RDI.

There is also no evidence to support the idea that apple cider vinegar makes it easier to absorb calcium.

On the good side, like all vinegars, it has virtually no kilojoules and, mixed with extra virgin olive oil, makes an excellent salad dressing.

Finally, a word of warning: don’t drink apple cider vinegar “neat”. It can damage the throat and oesophagus. Even diluted, its acidity can damage tooth enamel.

Daily Baby Aspirin May Lower Ovarian Cancer Risk

Daily Baby Aspirin May Lower Ovarian Cancer Risk

A standard aspirin did not lower ovarian cancer risk.

  • Taking low-dose aspirin is a daily routine for many people because it may lower the risk for heart attacks and strokes, and some research has tied it to a lower risk of colorectal cancer. Now a new study in JAMA Oncology suggests it may lower the risk for ovarian cancer as well.

Researchers followed more than 200,000 women for more than 25 years, recording data on lifestyle, health factors and disease outcomes and updating information with periodic interviews.

They found 1,054 cases of ovarian cancer. After adjusting for other variables, they found that women who took a baby aspirin — 100 milligrams or less — had a 23 percent reduced risk for ovarian cancer compared with those who did not. They found no risk reduction for those who took a standard 325-milligram pill.

“Our study has limits,” said the lead author, Mollie E. Barnard, a postdoctoral fellow at the Huntsman Cancer Institute at the University of Utah, “but we do have prospective data, and a very large sample followed over a long period of time. And we were able to look at standard and low-dose separately.”

How obesity causes cancer, and may make screening and treatment harder

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How obesity causes cancer, and may make screening and treatment harder

March 20, 2017 6.26am AEDT

Today, almost two in every three Australian adults are overweight or obese, as is one in four children. Obesity is a disease itself and a risk factor linked to ischaemic heart disease (the leading cause of premature deaths today in Australia), stroke (the third leading cause), and musculoskeletal conditions (the second major cause of disability), among others.

This rising obesity burden is the outcome of a host of factors, many of which are beyond the control of the individual. It is having a devastating impact on the health of the nation. What’s often overlooked though, is the link between obesity and cancer.

Cancer is a disease of altered gene expression that originates from changes to the DNA caused by a range of factors. These include inherited mutations, DNA damage, inflammation, hormones, and external factors including tobacco use, infections (for example viruses such as HPV), radiation, chemicals, and carcinogenic agents in food.

Strong evidence also links obesity to a number of cancers. These include oesophageal adenocarcinoma; bowel cancer (the third leading cause of preventable death in Australia); cancer of the liver, gall bladder and bile ducts; pancreatic cancer; postmenopausal breast cancer; endometrial cancer; kidney cancer; and multiple myeloma (cancer in the plasma in the blood).

This is just the tip of the iceberg, as highly suggestive evidence exists for a further eight cancers.

How does obesity increase the risk of cancer?

There are many complex ways obesity is thought to cause or increase the risk of cancer.

Increased body fat is associated with increased inflammation in the body, increased release of oestrogens (in part from the fat cells themselves), and decreased insulin sensitivity associated with raised insulin production.

Insulin, “insulin-like growth factor-1” (IGF1) and leptin are all elevated in obese people, and can promote the growth of cancer cells.

Secretion of the hormone insulin is usually tightly controlled and a healthy part of our body’s sugar regulation processes. But it can be significantly elevated in people with obesity-related pre-diabetes or diabetes due to insulin resistance.

This state of elevated insulin levels in the blood can act as a growth signal for tumour cells, and increases the risk of cancers of the colon and endometrium (the lining of the uterus), and likely of the pancreas and kidney.

Insulin-like growth factors (IGFs) regulate cell growth, differentiation and death, and IGF-1 has been associated with prostate, breast and bowel cancers.

Leptin, a hormone implicated in hunger and satiety, can stimulate proliferation of many pre-cancer and cancer cells. Increased leptin levels in obese people are associated with bowel and prostate cancers.

Sex steroid hormones including oestrogens, testosterone, and progesterone are crucial to healthy body development and sexual function, but are also likely to play a role in obesity and cancer. Increased levels of sex steroids are strongly associated with risk of developing endometrial and postmenopausal breast cancers, and may contribute to other cancers such as bowel cancer.

Fat tissue is the main site of oestrogen production in the body for men and postmenopausal women (while in premenopausal women the ovaries are the major producer). Obesity can predispose premenopausal women to polycystic ovarian syndrome, which causes elevated testosterone and therefore could contribute to cancer risk.

Obesity also causes inflammation in the body, meaning the body’s immune system is consistently more active than is normal in healthy weight people.

Evidence for a role of sex hormones and chronic inflammation in affecting the relationship between obesity and cancer is strong, and the evidence for a role of insulin and IGF is moderate. There are a range of other mechanisms still under investigation.

Where does obesity lie on cancer-risk scale?

Overall, obesity-associated cancers represent up to 8.2% of all cancers in the UK, compared to smoking which is responsible for approximately 19%.

Of all deaths from cancer in the USA, excess body weight is close behind smoking as the attributable cause, at 20% versus 30% respectively.

Does obesity affect the screening and detection of cancer?

Focusing on just two types of cancer, breast cancer in women and prostate cancer in men, some evidence suggests that obesity can delay the identification of cancer through screening – but does not reduce the importance or accuracy of screening tools or programs.

For breast cancer, the most common form of cancer in women in Australia, the good news is that screening accuracy is similar across weight status. The Swiss national health survey found the accuracy of mammography is maintained in obese women – with similar ability of the tests to detect cancers, but reduced ability to ensure the positive result definitely means cancer. This meant obese women had a 20% higher false positive rate than normal weight individuals, but does not suggest any cancers were missed.

The troubling news though is, studies suggest obese women with breast cancer detected through mammogram tend to present to their doctors later, and when the cancer is more serious, than their healthy weight counterparts. The exact reasons for this are not clear but may include possible difficulties in breast self-examination and delayed health-seeking. Such findings reinforce the crucial importance of strategies to encourage appropriate cancer screening and timely medical follow-up among overweight and obese women.

For prostate cancer, the most common form of cancer in Australia, large studies suggest a link between obesity and decreased risk of low-grade or early prostate cancer, but increased risk of advanced disease.

The reasons for this are again thought to be numerous, but one potential reason may be linked to greater difficulty in diagnosing prostate cancer in overweight men. While this is thought to possibly delay diagnosis and treatment, it is unlikely entirely to explain the links between obesity and prostate cancer risk.

What risks does obesity pose in the treatments of cancer?

Obesity can impact cancer treatments and their success. Obese patients have a significantly higher risk of heart attack following surgery, as well as risk of wound infection, nerve injury, and urinary infection. Obesity alone increases the risk of poorer health outcomes following surgery, and morbid obesity increases the risk of death.

In cancer treatments, one study has shown significantly increased surgical complications and prolonged hospital stay with morbid obesity in bowel cancer. Another suggests obesity may reduce chemotherapy efficacy in breast cancer, with lower disease-free survival rates.

Is this risk reversible?

By 2025 it’s estimated that more Australians will be obese than normal weight. At the same time, cancer is a leading contributor to early deaths and disability in Australia and the major cause of years lost from people’s lifespans.

The question is not whether obesity can cause cancer; it is how we can better prevent or mitigate this important risk factor. Reassuringly, there is suggestive evidence that weight loss may reduce or reverse many of the above processes and their associated risks.

While obesity is just one of the drivers of the cancer burden in Australia, it is one that is preventable and in doing so, would bring other enormous health benefits

Decreased mortality risk due to first acute coronary syndrome in women with postmenopausal hormone therapy use

A point I have stressed over the years is that hormone treatment in the menopause is good for your heart. It will reduce the risk of a heart attack. So when we consider that heart disease kill most women over 50 (far more than die from breast cancer), it is a very important finding. I have published many other studies over the years showing the protective effect of oestrogen on women’s cardiovascular system in the menopause.

Decreased mortality risk due to first acute coronary syndrome in women with postmenopausal hormone therapy use – <!– [if IE 6]> /wro/ayds~ie6.js<![endif]–>

Objectives

The role of postmenopausal hormone therapy (HT) in the incidence of acute coronary syndrome (ACS) has been studied extensively, but less is known of the impact of HT on the mortality risk due to an ACS.

 

Study design and main outcome measures

We extracted from a population-based ACS register, FINAMI, 7258 postmenopausal women with the first ACS. These data were combined with HT use data from the National Drug Reimbursement Register; 625 patients (9%) had used various HT regimens. The death risks due to ACS before admission to hospital, 2–28, or 29–365 days after the incident ACS were compared between HT users and non-users with logistic regression analyses.

Results

In all follow-up time points, the ACS death risks in HT ever-users were smaller compared to non-users. Of women with HT ever use, 42% died within one year as compared with 52% of non-users (OR 0.62, p < 0.001). Most deaths (84%) occurred within 28 days after the ACS, and in this group 36% of women with ever use of HT (OR 0.73, p = 0.002) and 30% of women with ≥5 year HT use (OR 0.54, p < 0.001) died as compared to 43% of the non-users. Age ≤60 or >60 years at the HT initiation was accompanied with similar reductions in ACS mortality risk.

Conclusions

Postmenopausal HT use is accompanied with reduced mortality risk after primary ACS.

Dealing with anxiety during a cancer prognosis

Dealing with anxiety during a cancer prognosis

Posted by Andreas Obermair on 23 February 2017 | 0 Comments

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Anxiety is a common feeling that patients sometimes have when coping with upcoming surgery or even the possibility of a cancer diagnosis.  Seeing medical specialists, going through tests and having cancer treatment can be stressful. Some patients living with cancer have a low level of distress, whilst others can be highly distressed. During cancer diagnosis and treatment, patients may feel a loss of control, be fearful, panicked, or uncertain. They are not unusual responses to the upheaval of cancer.

In contrast to the adjustment to surgery or a potentially life-threatening condition, anxiety can also be a condition that has been present for a very long time. During the normal daily life, anxiety can be managed reasonably well; however, with additional stressors, patients lack options to handle these new stressors. In such cases anxiety can suddenly have profound effects on the health of patients needing the input of a gynaecological oncologist.

Our physical and mental health are connected. Poor physical health can affect you mentally, and poor mental health can affect you physically. In my almost 20 year experience, I find that patients with severe anxiety that is left untreated face a worse prognosis than patients with no anxiety or where the anxiety is treated.

It is important for our practice to find ways of helping you to deal with your feelings. Here I discuss some ways that may help reduce anxiety during cancer.

  • Find other patients to talk to who are going through the same journey. In my office, we can arrange contact between patients, we can also do this for patients who require certain operations that don’t involve cancer. It can greatly help someone when you can share your experiences and ways of coping with someone in a similar situation as you.
  • Develop relaxation methods. Breathing, mindfulness, or yoga. Try deep breathing and relaxation exercises several times a day. Researchers reviewed seven clinical trials which overall included 888 cancer patients and found that mindfulness-based interventions effectively relieved anxiety and depression among patients. Another review of studies found women with a breast cancer diagnosis who practiced yoga improved their mental health.
  • A balanced and nutritious diet will help you to keep as well as possible.
  • Exercise.  Regular exercise can be a healthy distraction. Exercise produces endorphins (chemicals in the brain that act as natural painkillers) and also improve the ability to sleep, which reduces stress. Physical exercise has attracted increased interest in rehabilitation of cancer patients. Particularly for patients undergoing chemotherapy, exercise can reduce the number and severity of physical and psychosocial treatment-related side effects and improve a patients’ quality of life. I am currently involved in the ECHO trial. The ECHO trial evaluates the effects of an exercise intervention during firstline chemotherapy for ovarian cancer. For those who may be interested in participating in the trial see here
  • See a GP with a special interest in anxiety. I do not prescribe medication for anxiety because it is outside the spectrum of my qualification. I witnessed that for some patients medication has worked wonders. They are so more relaxed and are able to tolerate treatment a lot better and enjoy life again. Think about also asking your GP to refer you to a psychologist who can work with you and your family for ongoing help.
  • Beyond Blue also has a 24-hour anonymous phone service for those suffering mental distress. The Cancer Council (13 11 20) helpline is a free, confidential telephone information and support service for cancer patients.

Anxiety is not a condition that can simply go away easily, and like all forms of therapy, the effect can vary.

Anxiety, if not addressed properly can affect cancer diagnosis and treatment in many ways. Almost never patients decline or request procedures acknowledging that they are anxious. Some patients may decline certain diagnostic procedures. This means that the information collected from a blood test or CT scan may be lost.

Other patients request procedures because they are anxious.

The above strategies are a good starting point to controlling anxiety, and after trying some different options you will find what works for you.

If you wish to receive regular information, tips, resources, reassurance and inspiration for up-to-date care, that is safe and sound and in line with latest research please subscribe here to receive my blog, or like Dr Andreas Obermair on Facebook. Should you find this article interesting, please feel free to share it.

Alcohol increases cancer risk, but don’t trust the booze industry to give you the facts straight

Alcohol increases cancer risk, but don’t trust the booze industry to give you the facts straight

The alcohol industry has been misleading the public about the real link between alcohol and cancer, with tactics similar to those used by the tobacco industry. This is the finding from research published today in the Drug and Alcohol Review journal. The misleading information is being passed off as health messaging to a public largely unaware that it’s coming from the industry itself.

For nearly three decades, we have known cancer risk increases with alcohol consumption levels, on a dose-to-response basis. That is, the more we drink the greater the risk.

Back in 1988, the World Health Organisation’s cancer research arm concluded that alcohol consumption was a group one carcinogen – a direct cause of cancer in humans. In Australia in 2010, about 3,200 cases of cancer were attributable to alcohol consumption.

And it doesn’t matter if you’re swilling shiraz at the Mosman Park Dinner Society or downing lager at the Bullamakanka Darts Club, it’s the alcohol, not the type of beverage, that does the damage.


Read more: Does alcohol cause cancer?


Led by the London School of Hygiene and Tropical Medicine, the latest study concluded:

The [alcohol industry] appears to be engaged in the extensive misrepresentation of evidence about the alcohol-related risk of cancer. These activities have parallels with those of the tobacco industry.

The authors pointed to three main strategies the industry uses to do this:

  • denying, omitting or disputing the evidence that alcohol consumption increases cancer risk
  • distorting the evidence by mentioning cancer but misrepresenting the link, such as implying risk is present only with higher levels of drinking
  • distracting or diverting discussion away from the independent effects of alcohol on common cancers, such as mentioning alcohol is one of many causes. Breast and bowel cancer appeared to be particular targets for this type of misrepresentation.

Dodgy public health messages?

The study assessed key messages provided by 27 organisations run, funded or controlled by the alcohol industry in English-speaking countries. Known as social aspects and public relations organisations, these bodies seek to shape consumer messaging about alcohol and seem to be gaining increasing resources and prominence.

One example of such an entity is the International Alliance for Responsible Drinking. The research paper states that its members include some of the world’s leading beer, wine and spirits producers.

Industry messages often distort alcohol’s link to cancer. Photo by Artem Pochepetsky on Unsplash

The Australian example included in the study is DrinkWise. Its 13-member board includes six alcohol industry representatives from groups such as the Distilled Spirits Industry Council of Australia, the Brewers Association of Australia and New Zealand and the Australian Hotels Association. DrinkWise says its “primary focus is to help bring about a healthier and safer drinking culture in Australia”.

A recent survey showed that, of the one in two people who had heard of DrinkWise, only around a third (37%) knew the alcohol industry funded it.

Drinkwise’s message on alcohol’s impact on cancer opens by saying alcohol is “one of a number of factors that contribute to the risk for developing certain types of cancer”. This is true. But some might consider this “distraction”.

The second message is:

Cancer risk associated with the consumption of alcohol is related to patterns of drinking, particularly heavy drinking over extended periods of time.

Again, technically true. But it avoids the fact low levels of consumption lead to low levels of risk increase. The research out today classified this as “distortion”.


Learn more about cancer risk: Interactive body map


The way information is framed is essential to the message people take from it. And there’s little doubt the alcohol industry are masters of communication.

Information such as that given by DrinkWise may invite a drinker looking to avoid the thought their own drinking might increase their risk of cancer to think: “but other things are more important causes of cancer” and “I am not a heavy drinker over a long period of time so I am OK”. Conveniently, “heavy drinker” is not clearly defined.

Too few people know alcohol is a significant contributor to cancer, and this is a problem.

Not enough know the link

A survey conducted in 2008 in Western Australia found over 55% of adult women, when prompted, recognised a link between alcohol consumption and cancer risk. In the same survey, around 42% of women reported believing red wine consumption helped to prevent cancer.

A more recent national survey found 53% of adults linked alcohol with cancer risk. And funnily enough, just over half (52%) of those drinking at high or very high risk levels did not perceive their drinking to be harmful.


Read more: Social acceptance of alcohol allows us to ignore its harms


In 2010-11, the WA government ran an education campaign to show how alcohol increased the risk of breast cancer. This improved community awareness of the link from 62% to 87%. It also led to more women reporting the intention to reduce their drinking.

Before the campaign, 10% of those who drank fewer than two standard drinks per day reported they intended to reduce drinking. This rose to 25% after seeing the campaign. Among those who drank more than two drinks a day, the figure went from 28% to 38.5%.

If these effects play out in the wider community through a national campaign, it may well result in a meaningful reduction in alcohol sales. Obviously, this is not in the industry’s interest.

Cancer is the most feared illness. This is particularly so for middle-aged and older people who see their peers increasingly getting cancer diagnoses. And yet new research shows people in the baby boomer generation are the group least likely to be reducing their consumption.

Research on health warnings on alcohol labels suggests messages about the link between booze and bowel cancer have the greatest effect on making drinkers think twice. So should we be surprised the industry selling the product is keen to muddy the waters about what is a clear connection between drinking and cancer?

For too many people, the alcohol and cancer story is new news. The more we drink the more we increase the risk of cancer.

This is another inconvenient truth to add to the list. But we must all have the information to allow us to make the choice. And the alcohol industry is clearly not the best source of that information.